IL-27: a potential biomarker for responders to glatiramer acetate therapy-Reference-Cited by-同舟云学术

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IL-27: a potential biomarker for responders to glatiramer acetate therapy

Published:2017-03 Issue: Volume:304 Page:21-28
ISSN:0165-5728
Container-title:Journal of Neuroimmunology
language:en
Short-container-title:Journal of Neuroimmunology

Author:

Mindur John E.,Valenzuela Reuben M.,Yadav Sudhir K.,Boppana Sridhar,Dhib-Jalbut Suhayl,Ito Kouichi

Funder

Teva Pharmaceutical Industries Ltd.

Publisher

Elsevier BV

Subject

Clinical Neurology,Neurology,Immunology,Immunology and Allergy

Reference64 articles.

1. Copolymer 1 induces T cells of the T helper type 2 that crossreact with myelin basic protein and suppress experimental autoimmune encephalomyelitis;Aharoni;Proc. Natl. Acad. Sci. U. S. A.,1997

2. Bystander suppression of experimental autoimmune encephalomyelitis by T cell lines and clones of the Th2 type induced by copolymer 1;Aharoni;J. Neuroimmunol.,1998

3. Specific Th2 cells accumulate in the central nervous system of mice protected against experimental autoimmune encephalomyelitis by copolymer 1;Aharoni;Proc. Natl. Acad. Sci. U. S. A.,2000

4. Glatiramer acetate-specific T cells in the brain express T helper 2/3 cytokines and brain-derived neurotrophic factor in situ;Aharoni;Proc. Natl. Acad. Sci. U. S. A.,2003

5. MHC class II signal transduction in human dendritic cells induced by a natural ligand, the LAG-3 protein (CD223);Andreae;Blood,2003

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