Glatiramer Acetate administration does not reduce damage after cerebral ischemia in mice-Reference-Cited by-同舟云学术

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Glatiramer Acetate administration does not reduce damage after cerebral ischemia in mice

Published:2013-01 Issue:1-2 Volume:254 Page:55-62
ISSN:0165-5728
Container-title:Journal of Neuroimmunology
language:en
Short-container-title:Journal of Neuroimmunology

Author:

Poittevin Marine,Deroide Nicolas,Azibani Feriel,Delcayre Claude,Giannesini Claire,Levy Bernard I.,Pocard Marc,Kubis Nathalie

Publisher

Elsevier BV

Subject

Clinical Neurology,Neurology,Immunology,Immunology and Allergy

Reference39 articles.

1. Copolymer 1 induces T cells of the T helper type 2 that crossreact with myelin basic protein and suppress experimental autoimmune encephalomyelitis;Aharoni;Proc. Natl. Acad. Sci.,1997

2. Specific Th2 cells accumulate in the central nervous system of mice protected against experimental autoimmune encephalomyelitis by copolymer 1;Aharoni;Proc. Natl. Acad. Sci.,2000

3. Glatiramer acetate-specific T cells in the brain express T helper 2/3 cytokines and brain-derived neurotrophic factor in situ;Aharoni;Proc. Natl. Acad. Sci.,2003

4. Neurogenesis and neuroprotection induced by peripheral immunomodulatory treatment of experimental autoimmune encephalomyelitis;Aharoni;J. Neurosci.,2005

5. The immunomodulator glatiramer acetate augments the expression of neurotrophic factors in brains of experimental autoimmune encephalomyelitis mice;Aharoni;Proc. Natl. Acad. Sci.,2005

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