The role of complement and complement regulators in mediating motor nerve terminal injury in murine models of Guillain–Barré syndrome-Reference-Cited by-同舟云学术

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The role of complement and complement regulators in mediating motor nerve terminal injury in murine models of Guillain–Barré syndrome

Published:2008-09 Issue: Volume:201-202 Page:172-182
ISSN:0165-5728
Container-title:Journal of Neuroimmunology
language:en
Short-container-title:Journal of Neuroimmunology

Author:

Willison Hugh J.,Halstead Susan K.,Beveridge Erin,Zitman Femke M.P.,Greenshields Kay N.,Morgan B. Paul,Plomp Jaap J.

Publisher

Elsevier BV

Subject

Neurology (clinical),Neurology,Immunology,Immunology and Allergy

Reference55 articles.

1. Lack of effect of Miller Fisher sera/plasmas on transmitter release from PC12 cells;Benatar;J. Neuroimmunol.,1997

2. Combined pre- and postsynaptic action of IgG antibodies in Miller Fisher syndrome;Buchwald;Neurology,2001

3. Complex gangliosides at the neuromuscular junction are membrane receptors for autoantibodies and botulinum neurotoxin but redundant for normal synaptic function;Bullens;J. Neurosci.,2002

4. Serum IgG antibody to ganglioside GQ1b is a possible marker of Miller Fisher syndrome;Chiba;Ann. Neurol.,1992

5. The mouse complement regulator CD59b is significantly expressed only in testis and plays roles in sperm acrosome activation and motility;Donev;Mol. Immunol.,2008

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