A novel rare variant of CNPY3 from familial NMOSD impairs the TLR-mediated immune response-Reference-Cited by-同舟云学术

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A novel rare variant of CNPY3 from familial NMOSD impairs the TLR-mediated immune response

Published:2023-04 Issue: Volume:377 Page:578065
ISSN:0165-5728
Container-title:Journal of Neuroimmunology
language:en
Short-container-title:Journal of Neuroimmunology

Author:

Mo Yongxin,Wang Shisi,Chang Yanyu,Sun Xiaobo,Liu Zheng,Sun Ping,Xu Yan,Zhong Xiaofen,Peng Lisheng

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Neurology (clinical),Neurology,Immunology,Immunology and Allergy

Reference23 articles.

1. High in vitro immune reactivity to Escherichia coli in neuromyelitis optica patients is correlated with both neurological disabilities and elevated plasma lipopolysaccharide levels;Barros;Hum. Immunol.,2013

2. Expansion of IL-6(+) Th17-like cells expressing TLRs correlates with microbial translocation and neurological disabilities in NMOSD patients;Barros;J. Neuroimmunol.,2017

3. Clinical and genetic analysis of familial neuromyelitis optica spectrum disorder in Chinese: associated with ubiquitin-specific peptidase USP18 gene variants;Chang;J. Neurol. Neurosurg. Psychiatry.,2022

4. Neuropeptides as targets for the development of anticonvulsant drugs;Clynen;Mol. Neurobiol.,2014

5. Cellular organization of glycosylation;Colley,2015

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Genetic Basis of Inflammatory Demyelinating Diseases of the Central Nervous System: Multiple Sclerosis and Neuromyelitis Optica Spectrum;Genes;2023-06-23

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