Genetic deletion of c-Jun amino-terminal kinase 3 (JNK3) modestly increases disease severity in a mouse model of multiple sclerosis-Reference-Cited by-同舟云学术

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Genetic deletion of c-Jun amino-terminal kinase 3 (JNK3) modestly increases disease severity in a mouse model of multiple sclerosis

Published:2023-09 Issue: Volume:382 Page:578152
ISSN:0165-5728
Container-title:Journal of Neuroimmunology
language:en
Short-container-title:Journal of Neuroimmunology

Author:

Priego Mercedes,Noriega Lorena,Kalinin Sergey,Hoffman Lisa M.,Feinstein Douglas L.,Morfini Gerardo

Publisher

Elsevier BV

Subject

Neurology (clinical),Neurology,Immunology,Immunology and Allergy

Reference47 articles.

1. JNK3 maintains expression of the insulin receptor substrate 2 (IRS2) in insulin-secreting cells: functional consequences for insulin signaling;Abdelli;PLoS One,2012

2. JNK3 is abundant in insulin-secreting cells and protects against cytokine-induced apoptosis;Abdelli;Diabetologia.,2009

3. Immune cell contributors to the female sex Bias in multiple sclerosis and experimental autoimmune encephalomyelitis;Alvarez-Sanchez;Curr. Top. Behav. Neurosci.,2023

4. C-Jun N-terminal kinase as a therapeutic target in experimental autoimmune encephalomyelitis;Bagnoud;Cells.,2020

5. Distinct roles of c-Jun N-terminal kinase isoforms in neurite initiation and elongation during axonal regeneration;Barnat;J. Neurosci.,2010

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