Growth associated protein (GAP-43): Cloning and the development of a sensitive ELISA for neurological disorders-Reference-Cited by-同舟云学术

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Growth associated protein (GAP-43): Cloning and the development of a sensitive ELISA for neurological disorders

Published:2014-11 Issue:1-2 Volume:276 Page:18-23
ISSN:0165-5728
Container-title:Journal of Neuroimmunology
language:en
Short-container-title:Journal of Neuroimmunology

Author:

Gnanapavan Sharmilee,Yousaf Nasim,Heywood Wendy,Grant Donna,Mills Kevin,Chernajovsky Yuti,Keir Geoff,Giovannoni Gavin

Publisher

Elsevier BV

Subject

Neurology (clinical),Neurology,Immunology,Immunology and Allergy

Reference28 articles.

1. Overexpression of the neural growth-associated protein GAP-43 induces nerve sprouting in the adult nervous system of transgenic mice;Aigner;Cell,1995

2. GAP-43: an intrinsic determinant of neuronal development and plasticity;Benowitz;Trends Neurosci.,1997

3. Anatomical distribution of the growth-associated protein GAP-43/B-50 in the adult rat brain;Benowitz;J. Neurosci.,1988

4. Localization of the growth-associated phosphoprotein GAP-43 (B-50, F1) in the human cerebral cortex;Benowitz;J. Neurosci.,1989

5. Reduced GAP-43 message levels are associated with increased neurofibrillary tangle density in the frontal association cortex (area 9) in Alzheimer's disease;Coleman;Neurobiol. Aging,1992

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