Author:
Borlongan Cesario V,Nishino Hitoo,Sanberg Paul R
Subject
General Medicine,General Neuroscience
Reference26 articles.
1. Beal, M.F., 1992. Does impairment of energy metabolism result in excitotoxic neruonal death in neruodegnerative illnesses? Ann. Neurol. 31, 119–130.
2. Beal, M.F., Brouillet, E., Jenkins, B., Ferrante, R.J., Kowall, N.W., Miller, J.M., Storey, E., Srivastava, R., Rosen, B.R., Hyman, B.T., 1993. Neurochemical and histologic characterization of striatal excitotoxic lesions produced by the mitochondrial toxin 3-nitropropionic acid. J. Neurosci. 13, 1481–1492.
3. Borlongan, C.V., Koutouzis, T.K., Freeman, T.B., Cahill, D.W., Sanberg, P.R., 1995. Systemic 3-nitropropionic aicd: behavioral deficits and striatal damage in rats. Brain Res. Bull. 36, 549–556.
4. Borlongan, C.V., Koutouzis, T.K., Freeman, T.B., Cahill, D.W., Sanberg, P.R., 1996a. Behavioral pathology induced by repeated systemic injections of 3-nitropropionic acid mimics the motoric symptoms of Huntington's disease. Brain Res. 697, 254–257.
5. Borlongan, C.V., Koutouzis, T.K., Poulos, S., Saporta, S., Freeman, T.B., Cahill, D.W., Sanberg, P.R., 1996b. Bilateral fetal striatal transplantation in the 3-nitropropionic acid-induced hypoactive model of Huntington's disease. Soc. Neurosci. Abstr. 22, 580.
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