CDKN2A-p16 Deletion and Activated KRASG12D Drive Barrett’s-Like Gland Hyperplasia-Metaplasia and Synergize in the Development of Dysplasia Precancer Lesions-Reference-Cited by-同舟云学术

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CDKN2A-p16 Deletion and Activated KRASG12D Drive Barrett’s-Like Gland Hyperplasia-Metaplasia and Synergize in the Development of Dysplasia Precancer Lesions

Published:2024 Issue:5 Volume:17 Page:769-784
ISSN:2352-345X
Container-title:Cellular and Molecular Gastroenterology and Hepatology
language:en
Short-container-title:Cellular and Molecular Gastroenterology and Hepatology

Author:

Sun Jing,Sepulveda Jorge L.,Komissarova Elena V.,Hills Caitlin,Seckar Tyler D.,LeFevre Narine M.,Simonyan Hayk,Young Colin,Su Gloria,Del Portillo Armando,Wang Timothy C.,Sepulveda Antonia R.^ORCID

Funder

George Washington University School of Medicine and Health Sciences

Publisher

Elsevier BV

Reference45 articles.

1. Evaluation of mutational testing of preneoplastic Barrett’s mucosa by next-generation sequencing of formalin-fixed, paraffin-embedded endoscopic samples for detection of concurrent dysplasia and adenocarcinoma in Barrett’s esophagus;Del Portillo;J Mol Diagn,2015

2. Exome and whole-genome sequencing of esophageal adenocarcinoma identifies recurrent driver events and mutational complexity;Dulak;Nat Genet,2013

3. High-resolution genomic alterations in Barrett’s metaplasia of patients who progress to esophageal dysplasia and adenocarcinoma;Sepulveda;Int J Cancer,2019

4. Diagnosis and management of Barrett’s esophagus: an updated ACG guideline;Shaheen;Am J Gastroenterol,2022

5. Global burden and epidemiology of Barrett oesophagus and oesophageal cancer;Thrift;Nat Rev Gastroenterol Hepatol,2021

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