Critical role of c-jun N-terminal protein kinase in promoting mitochondrial dysfunction and acute liver injury-Reference-Cited by-同舟云学术

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Critical role of c-jun N-terminal protein kinase in promoting mitochondrial dysfunction and acute liver injury

Published:2015-12 Issue: Volume:6 Page:552-564
ISSN:2213-2317
Container-title:Redox Biology
language:en
Short-container-title:Redox Biology

Author:

Jang Sehwan,Yu Li-Rong^ORCID,Abdelmegeed Mohamed A.,Gao Yuan,Banerjee Atrayee,Song Byoung-Joon

Funder

National Institute of Alcohol Abuse and Alcoholism

National Center for Toxicological Research

U.S. Food and Drug Administration

Publisher

Elsevier BV

Subject

Organic Chemistry,Biochemistry,Clinical Biochemistry

Reference59 articles.

1. Acetaminophen and the U.S. acute liver failure study group: lowering the risks of hepatic failure;Lee;Hepatology,2004

2. Pathogenesis of liver injury in acute liver failure;Chung;Gastroenterology,2012

3. Potentiation of acetaminophen hepatotoxicity by alcohol;McClain;JAMA,1980

4. Acetaminophen hepatotoxicity in alcoholics;Seeff;Ann. Int. Med.,1986

5. Mechanisms of carbon tetrachloride toxicity;Recknagel;Pharmacol. Ther.,1989

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