Alexander disease GFAP R239C mutant shows increased susceptibility to lipoxidation and elicits mitochondrial dysfunction and oxidative stress-Reference-Cited by-同舟云学术

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Alexander disease GFAP R239C mutant shows increased susceptibility to lipoxidation and elicits mitochondrial dysfunction and oxidative stress

Published:2022-09 Issue: Volume:55 Page:102415
ISSN:2213-2317
Container-title:Redox Biology
language:en
Short-container-title:Redox Biology

Author:

Viedma-Poyatos Álvaro,González-Jiménez Patricia,Pajares María A.,Pérez-Sala Dolores^ORCID

Funder

Agencia Estatal de Investigación

Family Process Institute

Instituto de Salud Carlos III

European Regional Development Fund

Ministerio de Ciencia e Innovación

Publisher

Elsevier BV

Subject

Organic Chemistry,Biochemistry,Clinical Biochemistry

Reference88 articles.

1. GFAP and its role in Alexander disease;Quinlan;Exp. Cell Res.,2007

2. Glial fibrillary acidic protein (GFAP) and the astrocyte intermediate filament system in diseases of the central nervous system;Hol;Curr. Opin. Cell Biol.,2015

3. Alexander disease;Messing;Handb. Clin. Neurol.,2018

4. Alexander's disease: clinical, pathologic, and genetic features;Johnson;J. Child Neurol.,2003

5. Intermediate filament protein partnership in astrocytes;Eliasson;J. Biol. Chem.,1999

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