Disrupting the vicious cycle created by NOX activation in sickle erythrocytes exposed to hypoxia/reoxygenation prevents adhesion and vasoocclusion-Reference-Cited by-同舟云学术

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Disrupting the vicious cycle created by NOX activation in sickle erythrocytes exposed to hypoxia/reoxygenation prevents adhesion and vasoocclusion

Published:2019-07 Issue: Volume:25 Page:101097
ISSN:2213-2317
Container-title:Redox Biology
language:en
Short-container-title:Redox Biology

Author:

MacKinney Anson,Woska Emily,Spasojevic Ivan,Batinic-Haberle Ines,Zennadi Rahima

Funder

National Institutes of Health

National Heart, Lung, and Blood Institute

Publisher

Elsevier BV

Subject

Organic Chemistry,Biochemistry,Clinical Biochemistry

Reference72 articles.

1. Monoclonal antibodies to alphaVbeta3 (7E3 and LM609) inhibit sickle red blood cell-endothelium interactions induced by platelet-activating factor;Kaul;Blood,2000

2. G6PD deficiency, absence of alpha-thalassemia, and hemolytic rate at baseline are significant independent risk factors for abnormally high cerebral velocities in patients with sickle cell anemia;Bernaudin;Blood,2008

3. Erythrocyte adherence to endothelium in sickle-cell anemia. A possible determinant of disease severity;Hebbel;N. Engl. J. Med.,1980

4. Dysregulated arginine metabolism, hemolysis-associated pulmonary hypertension, and mortality in sickle cell disease;Morris;JAMA: J. Am. Med. Assoc.,2005

5. Hemoglobin S polymerization: primary determinant of the hemolytic and clinical severity of the sickling syndromes;Brittenham;Blood,1985

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