Neonatal iron supplementation potentiates oxidative stress, energetic dysfunction and neurodegeneration in the R6/2 mouse model of Huntington's disease-Reference-Cited by-同舟云学术

Neonatal iron supplementation potentiates oxidative stress, energetic dysfunction and neurodegeneration in the R6/2 mouse model of Huntington's disease

Published:2015-04 Issue: Volume:4 Page:363-374
ISSN:2213-2317
Container-title:Redox Biology
language:en
Short-container-title:Redox Biology

Author:

Berggren Kiersten L.,Chen Jianfang,Fox Julia,Miller Jonathan,Dodds Lindsay,Dugas Bryan,Vargas Liset,Lothian Amber,McAllum Erin,Volitakis Irene,Roberts Blaine,Bush Ashley I.,Fox Jonathan H.

Funder

NINDS

Publisher

Elsevier BV

Subject

Organic Chemistry,Biochemistry,Clinical Biochemistry

Reference48 articles.

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3. Relationship between trinucleotide repeat expansion and phenotypic variation in Huntington’s disease;Snell;Nature Genetics,1993

4. Age, CAG repeat length, and clinical progression in Huntington’s disease;Rosenblatt;Movement Disorders: Official Journal of the Movement Disorder Society,2012

5. Genetic modifiers of Huntington’s disease: beyond CAG;Arning;Future Neurology,2012

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