Oxidative stress modulates rearrangement of endoplasmic reticulum-mitochondria contacts and calcium dysregulation in a Friedreich's ataxia model-Reference-Cited by-同舟云学术

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Oxidative stress modulates rearrangement of endoplasmic reticulum-mitochondria contacts and calcium dysregulation in a Friedreich's ataxia model

Published:2020-10 Issue: Volume:37 Page:101762
ISSN:2213-2317
Container-title:Redox Biology
language:en
Short-container-title:Redox Biology

Author:

Rodríguez Laura R.,Calap-Quintana Pablo,Lapeña-Luzón Tamara,Pallardó Federico V.,Schneuwly Stephan,Navarro Juan A.,Gonzalez-Cabo Pilar

Funder

Deutsche Forschungsgemeinschaft

Ministerio de Economía y Competitividad

Publisher

Elsevier BV

Subject

Organic Chemistry,Biochemistry,Clinical Biochemistry

Reference70 articles.

1. Friedreich's ataxia: autosomal recessive disease caused by an intronic GAA triplet repeat expansion;Campuzano;Science,1996

2. Clinical features of Friedreich's ataxia: classical and atypical phenotypes;Parkinson;J. Neurochem.,2013

3. Friedreich ataxia: neuropathology revised;Koeppen;J. Neuropathol. Exp. Neurol.,2013

4. The role of iron in friedreich's ataxia: insights from studies in human tissues and cellular and animal models;Llorens;Front. Neurosci.,2019

5. An interaction between frataxin and Isu1/Nfs1 that is crucial for Fe/S cluster synthesis on Isu1;Gerber;EMBO Rep.,2003

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