Mitochondrial TXNRD3 confers drug resistance via redox-mediated mechanism and is a potential therapeutic target in vivo-Reference-Cited by-同舟云学术

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Mitochondrial TXNRD3 confers drug resistance via redox-mediated mechanism and is a potential therapeutic target in vivo

Published:2020-09 Issue: Volume:36 Page:101652
ISSN:2213-2317
Container-title:Redox Biology
language:en
Short-container-title:Redox Biology

Author:

Liu Xiaoxia,Zhang Yanyu,Lu Wenhua,Han Yi,Yang Jing,Jiang Weiye,You Xin,Luo Yao,Wen Shijun,Hu Yumin,Huang Peng

Funder

National Key R&D Program of China

National Natural Science Foundation of China

Sun Yat-sen University

Publisher

Elsevier BV

Subject

Organic Chemistry,Biochemistry,Clinical Biochemistry

Reference45 articles.

1. A novel microtubule inhibitor overcomes multidrug resistance in tumors;Ning;Cancer Res.,2018

2. EGFR mutation and response of lung cancer to gefitinib;Toyooka;N. Engl. J. Med.,2005

3. Tumor cells can follow distinct evolutionary paths to become resistant to epidermal growth factor receptor inhibition;Hata;Nat. Med.,2016

4. Association of constitutively activated hepatocyte growth factor receptor (Met) with resistance to a dual EGFR/Her2 inhibitor in non-small-cell lung cancer cells;Agarwal;Br. J. Canc.,2009

5. Prolonged exposure to FLT3 inhibitors leads to resistance via activation of parallel signaling pathways;Piloto;Blood,2007

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