Knockout of cyclophilin D in Ppif−/− mice increases stability of brain mitochondria against Ca2+ stress-Reference-Cited by-同舟云学术

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Knockout of cyclophilin D in Ppif−/− mice increases stability of brain mitochondria against Ca2+ stress

Published:2015-08 Issue: Volume:579 Page:40-46
ISSN:0003-9861
Container-title:Archives of Biochemistry and Biophysics
language:en
Short-container-title:Archives of Biochemistry and Biophysics

Author:

Gainutdinov T.,Molkentin J.D.,Siemen D.,Ziemer M.,Debska-Vielhaber G.,Vielhaber S.,Gizatullina Z.,Orynbayeva Z.,Gellerich F.N.

Funder

Deutsches Zentrum für Neurodegenerative Erkrankungen

Drexel University Professional Enrichment and Growth

Publisher

Elsevier BV

Subject

Molecular Biology,Biochemistry,Biophysics

Reference51 articles.

1. Cyclophilin and peptidyl-prolyl cis-trans isomerase are probably identical proteins

2. Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

3. Cyclophilin D Deficiency Rescues Axonal Mitochondrial Transport in Alzheimer’s Neurons

4. Cyclophilin D deficiency improves mitochondrial function and learning/memory in aging Alzheimer disease mouse model

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