Severity and duration of hypoxic stress differentially regulates HIF-1α-mediated cardiomyocyte apoptotic signaling milieu during myocardial infarction

Author:

Datta Chaudhuri Ratul,Banerjee Durba,Banik Anirban,Sarkar Sagartirtha

Funder

Department of Science and Technology

Department of Biotechnology

MHRD-STARS

DST-FIST

University Grants Commission

Council of Scientific & Industrial Research

Publisher

Elsevier BV

Subject

Molecular Biology,Biochemistry,Biophysics

Reference68 articles.

1. Apoptotic and necrotic myocyte cell deaths are independent contributing variables of infarct size in rats;Kajstura;Lab. Invest. J. Tech. Methods Pathol.,1996

2. Role of α-crystallin B as a regulatory switch in modulating cardiomyocyte apoptosis by mitochondria or endoplasmic reticulum during cardiac hypertrophy and myocardial infarction;Mitra;Cell Death Dis.,2013

3. Intracellular signaling by reactive oxygen species during hypoxia in cardiomyocytes;Duranteau;J. Biol. Chem.,1998

4. Redox signaling in cardiac myocytes;Santos;Free Radic. Biol. Med.,2011

5. Regulation of hypoxia-inducible factor-1α by nitric oxide through mitochondria-dependent and-independent pathways;Mateo;Biochem. J.,2003

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