Amino acid residue E543 in JAK2 C618R is a potential therapeutic target for myeloproliferative disorders caused by JAK2 C618R mutation-Reference-Cited by-同舟云学术

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Amino acid residue E543 in JAK2 C618R is a potential therapeutic target for myeloproliferative disorders caused by JAK2 C618R mutation

Published:2012-12 Issue:1 Volume:528 Page:57-66
ISSN:0003-9861
Container-title:Archives of Biochemistry and Biophysics
language:en
Short-container-title:Archives of Biochemistry and Biophysics

Author:

Wu Qing-Yun,Li Feng,Guo Hua-Yan,Cao Jiang,Chen Chong,Chen Wei,Zhao Kai,Zeng Ling-Yu,Han Zheng-Xiang,Li Zhen-Yu,Wang Xiao-Yun,Xu Kai-Lin

Publisher

Elsevier BV

Subject

Molecular Biology,Biochemistry,Biophysics

Reference39 articles.

1. Getting to know JAK

2. Dimerization by a Cytokine Receptor Is Necessary for Constitutive Activation of JAK2V617F

3. The JAK/STAT signaling pathway

4. Signaling through the JAK/STAT pathway, recent advances and future challenges

5. Challenges Facing JAK Inhibitor Therapy for Myeloproliferative Neoplasms

Cited by 10 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Insights into the role of JAK2-I724T variant in myeloproliferative neoplasms from a unique cohort of New Zealand patients;Hematology;2024-01-10

2. Roles of T875N somatic mutation in the activity, structural stability of JAK2 and the transformation of OCI-AML3 cells;International Journal of Biological Macromolecules;2019-09

3. Disruption of R867 and Y613 interaction plays key roles in JAK2 R867Q mutation caused acute leukemia;International Journal of Biological Macromolecules;2019-09

4. The regulation of JAKs in cytokine signaling and its breakdown in disease;Cytokine;2019-06

5. Loss of K607 and E877 interaction is a key reason for JAK2 K607N mutation caused acute myeloid leukemia;International Journal of Biological Macromolecules;2019-03

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