Evolutionary conserved N-terminal domain of Nrf2 is essential for the Keap1-mediated degradation of the protein by proteasome-Reference-Cited by-同舟云学术

登录注册会员服务联系我们

Evolutionary conserved N-terminal domain of Nrf2 is essential for the Keap1-mediated degradation of the protein by proteasome

Published:2005-01 Issue:2 Volume:433 Page:342-350
ISSN:0003-9861
Container-title:Archives of Biochemistry and Biophysics
language:en
Short-container-title:Archives of Biochemistry and Biophysics

Author:

Katoh Yasutake,Iida Katsuyuki,Kang Moon-IL,Kobayashi Akira,Mizukami Mio,Tong Kit I.,McMahon Michael,Hayes John D.,Itoh Ken,Yamamoto Masayuki

Publisher

Elsevier BV

Subject

Molecular Biology,Biochemistry,Biophysics

Reference40 articles.

1. The electrophile counterattack response: Protection against neoplasia and toxicity

2. Antioxidant-Inducible Genes

3. Isolation of NF-E2-related factor 2 (Nrf2), a NF-E2-like basic leucine zipper transcriptional activator that binds to the tandem NF-E2/AP1 repeat of the beta-globin locus control region.

4. Cloning and characterization of a novel erythroid cell-derived CNC family transcription factor heterodimerizing with the small Maf family proteins.

5. An Nrf2/Small Maf Heterodimer Mediates the Induction of Phase II Detoxifying Enzyme Genes through Antioxidant Response Elements

Cited by 188 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Transcription factor NF-E2-related factor 2 plays a critical role in acute lung injury/acute respiratory distress syndrome (ALI/ARDS) by regulating ferroptosis;PeerJ;2024-07-30

2. Effect of RNF113A deficiency on oxidative stress-induced NRF2 pathway;Animal Cells and Systems;2024-05-11

3. Nuclear Factor Erythroid 2 Related Factor 2 and Mitochondria Form a Mutually Regulating Circuit in the Prevention and Treatment of Metabolic Syndrome;Antioxidants & Redox Signaling;2024-03-20

4. Disrupting the SKN-1 homeostat: mechanistic insights and phenotypic outcomes;Frontiers in Aging;2024-03-04

5. Broadening horizons: ferroptosis as a new target for traumatic brain injury;Burns & Trauma;2024-01-01

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号京ICP备18003416号-3