Involvement of cyclophilin D in mitochondrial permeability transition induction in intact cells
Author:
Publisher
Elsevier BV
Subject
Molecular Biology,Biochemistry,Biophysics
Reference24 articles.
1. Transient and Long-Lasting Openings of the Mitochondrial Permeability Transition Pore Can Be Monitored Directly in Intact Cells by Changes in Mitochondrial Calcein Fluorescence
2. Mitochondria Are Excitable Organelles Capable of Generating and Conveying Electrical and Calcium Signals
3. Permeability transition pore regulates both mitochondrial membrane potential and agonist-evoked Ca2+signals in oligodendrocyte progenitors
4. From calcium signaling to cell death: two conformations for the mitochondrial permeability transition pore. Switching from low- to high-conductance state
5. A pore way to die
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1. Long-term inhibition of cyclophilin D results in intracellular translocation of calcein AM from mitochondria to lysosomes;Archives of Biochemistry and Biophysics;2017-01
2. Partial contribution of mitochondrial permeability transition to t -butyl hydroperoxide-induced cell death;Biochemistry and Biophysics Reports;2016-09
3. CKMT1 regulates the mitochondrial permeability transition pore in a process that provides evidence for alternative forms of the complex;Journal of Cell Science;2014-04-15
4. KB-R7943, a plasma membrane Na+/Ca2+ exchanger inhibitor, blocks opening of the mitochondrial permeability transition pore;Biochemical and Biophysical Research Communications;2014-01
5. Cytotoxicity of surface-functionalized silicon and germanium nanoparticles: the dominant role of surface charges;Nanoscale;2013
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