DJ-1 activates autophagy in the repression of cardiac hypertrophy-Reference-Cited by-同舟云学术

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DJ-1 activates autophagy in the repression of cardiac hypertrophy

Published:2017-11 Issue: Volume:633 Page:124-132
ISSN:0003-9861
Container-title:Archives of Biochemistry and Biophysics
language:en
Short-container-title:Archives of Biochemistry and Biophysics

Author:

Xue Ruicong,Jiang Jingzhou,Dong Bin,Tan Weiping,Sun Yu,Zhao Jingjing,Chen Yili,Dong Yugang,Liu Chen

Funder

National Key R&D Program

National Natural Science Foundation of China

Guangdong Natural Science Foundation

Major Program of Guangdong Natural Science Foundation

Science and Technology Program Foundation of Guangdong Province

Publisher

Elsevier BV

Subject

Molecular Biology,Biochemistry,Biophysics

Reference39 articles.

1. Left ventricular hypertrophy: an overlooked cardiovascular risk factor;Bauml;Cleve Clin. J. Med.,2010

2. Progression from compensated hypertrophy to failure in the pressure-overloaded human heart: structural deterioration and compensatory mechanisms;Hein;Circulation,2003

3. The cellular and molecular response of cardiacmyocytes to mechanical stress;Sadoshima;Annu. Rev. Physiol.,1997

4. Autophagy in metazoans: cell survival in the land of plenty;Lum;Nat. Rev. Mol. Cell Biol.,2005

5. The role of autophagy in cardiomyocytes in the basal state and in response to hemodynamic stress;Nakai;Nat. Med.,2007

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1. Emerging markers of inflammation and oxidative stress as potential predictors of coronary artery disease;International Journal of Cardiology;2023-04

2. DJ-1 promotes energy balance by regulating both mitochondrial and autophagic homeostasis;Neurobiology of Disease;2023-01

3. DGKζ deficiency promotes maladaptive cardiac hypertrophy through modulating autophagy via mTOR/TFEB signaling pathway;2022-11-11

4. Disuse-associated loss of the protease LONP1 in muscle impairs mitochondrial function and causes reduced skeletal muscle mass and strength;Nature Communications;2022-02-16

5. DJ-1 activates the noncanonical NF-κB pathway via interaction with Cezanne to inhibit the apoptosis and promote the proliferation of Ishikawa cells;Molecular Biology Reports;2021-08

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