Passive Ca2+ overload in H9c2 cardiac myoblasts: Assessment of cellular damage and cytosolic Ca2+ transients-Reference-Cited by-同舟云学术

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Passive Ca2+ overload in H9c2 cardiac myoblasts: Assessment of cellular damage and cytosolic Ca2+ transients

Published:2011-08 Issue:2 Volume:512 Page:175-182
ISSN:0003-9861
Container-title:Archives of Biochemistry and Biophysics
language:en
Short-container-title:Archives of Biochemistry and Biophysics

Author:

Soler Fernando,Lax Antonio,Carmen Asensio M.,Pascual-Figal Domingo,Fernández-Belda Francisco

Funder

Spanish Ministerio de Educación y Cultura

Fundación Séneca Región de Murcia

Publisher

Elsevier BV

Subject

Molecular Biology,Biochemistry,Biophysics

Reference34 articles.

1. Cardiac excitation–contraction coupling

2. The versatility and universality of calcium signalling

3. The control of sarcoplasmic reticulum Ca content in cardiac muscle

4. Sarcoplasmic reticulum calsequestrins: Structural and functional properties

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1. Crocin, a carotenoid component of Crocus cativus, exerts inhibitory effects on L-type Ca2+ current, Ca2+ transient, and contractility in rat ventricular myocytes;Canadian Journal of Physiology and Pharmacology;2016-03

2. Early oxidative damage induced by doxorubicin: Source of production, protection by GKT137831 and effect on Ca2+ transporters in HL-1 cardiomyocytes;Archives of Biochemistry and Biophysics;2016-03

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