Supplement of TCA cycle intermediates protects against high glucose/palmitate-induced INS-1 beta cell death-Reference-Cited by-同舟云学术

Supplement of TCA cycle intermediates protects against high glucose/palmitate-induced INS-1 beta cell death

Published:2011-01 Issue:2 Volume:505 Page:231-241
ISSN:0003-9861
Container-title:Archives of Biochemistry and Biophysics
language:en
Short-container-title:Archives of Biochemistry and Biophysics

Author:

Choi Sung-E,Lee Youn-Jung,Hwang Geum-Sook,Chung Joo Hee,Lee Soo-Jin,Lee Ji-Hyun,Han Seung Jin,Kim Hae Jin,Lee Kwan-Woo,Kim Youngsoo,Jun Hee-Sook,Kang Yup

Publisher

Elsevier BV

Subject

Molecular Biology,Biochemistry,Biophysics

Reference56 articles.

1. Inflammatory mediators and islet ?-cell failure: a link between type 1 and type 2 diabetes

2. Glucolipotoxicity: Fuel Excess and β-Cell Dysfunction

3. Prolonged Exposure to Free Fatty Acids Has Cytostatic and Pro-Apoptotic Effects on Human Pancreatic Islets

4. Saturated Fatty Acids Synergize with Elevated Glucose to Cause Pancreatic β-Cell Death

5. Monounsaturated Fatty Acids Prevent the Deleterious Effects of Palmitate and High Glucose on Human Pancreatic β-Cell Turnover and Function

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