Endoplasmic reticulum stress-induced complex I defect: Central role of calcium overload-Reference-Cited by-同舟云学术

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Endoplasmic reticulum stress-induced complex I defect: Central role of calcium overload

Published:2020-04 Issue: Volume:683 Page:108299
ISSN:0003-9861
Container-title:Archives of Biochemistry and Biophysics
language:en
Short-container-title:Archives of Biochemistry and Biophysics

Author:

Mohsin Ahmed A.,Thompson Jeremy,Hu Ying,Hollander John,Lesnefsky Edward J.,Chen Qun

Funder

PDH

National Institute on Aging

Office of Research and Development

Medical Research Service Merit Review Award

Virginia Commonwealth University

National Institutes of Health

Community Foundation

Publisher

Elsevier BV

Subject

Molecular Biology,Biochemistry,Biophysics

Reference79 articles.

1. Mitochondrial metabolism in aging heart;Lesnefsky;Circ. Res.,2016

2. Mitochondrial dysfunction and myocardial ischemia-reperfusion: implications for novel therapies;Lesnefsky;Annu. Rev. Pharmacol. Toxicol.,2017

3. Crosstalk signaling between mitochondrial Ca2+ and ROS;Feissner;Front Biosci (Landmark Ed),2009

4. Bcl-2 family members and apoptosis, taken to heart;Gustafsson;Am. J. Physiol. Cell Physiol.,2007

5. Redox regulation of sodium and calcium handling;Wagner;Antioxidants Redox Signal.,2013

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4. Xingnaojing injection alleviates cerebral ischemia/reperfusion injury through regulating endoplasmic reticulum stress in Vivo and in Vitro;Heliyon;2024-02

5. Acute endoplasmic reticulum stress‐induced mitochondria respiratory chain damage: The role of activated calpains;The FASEB Journal;2024-01-10

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