Priming of 6-hydroxydopamine-lesioned rats with l-DOPA or quinpirole results in an increase in dopamine D1 receptor-dependent cyclic AMP production in striatal tissue-Reference-Cited by-同舟云学术

Priming of 6-hydroxydopamine-lesioned rats with l-DOPA or quinpirole results in an increase in dopamine D1 receptor-dependent cyclic AMP production in striatal tissue

Published:1997-07 Issue:1 Volume:331 Page:23-26
ISSN:0014-2999
Container-title:European Journal of Pharmacology
language:en
Short-container-title:European Journal of Pharmacology

Author:

Pinna Annalisa,Morelli Micaela,Drukarch Benjamin,Stoof Johannes C

Publisher

Elsevier BV

Subject

Pharmacology

Reference14 articles.

1. Homologous desensitization of the D1 dopamine receptor;Balmforth;J. Neurochem.,1990

2. Behavioral sensitization in 6-hydroxydopamine lesioned rats involves the dopamine signal transduction: Changes in DARPP-32 phosphorylation;Barone;Neuroscience,1994

3. Lack of a dopamine autoreceptor selective profile of B-HT 920 in functional in vitro model systems of D2 receptors in rat striatum;Drukarch;Eur. J. Pharmacol.,1990

4. Dopaminergic functional supersensitivity: Effects of chronic l-dopa and carbidopa treatment in an animal model of Parkinson's disease;Hossain;J. Pharmacol. Exp. Ther.,1993

5. Stimulation by dopamine of cyclic 3′,5′-monophosphate formation in rat caudate nucleus: Effect of lesions of the nigro-neostriatal pathway;Krueger;Mol. Pharmacol.,1976

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