Complement genetics and susceptibility to inflammatory disease. Lessons from genotype–phenotype correlations-Reference-Cited by-同舟云学术

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Complement genetics and susceptibility to inflammatory disease. Lessons from genotype–phenotype correlations

Published:2016-06 Issue:6 Volume:221 Page:709-714
ISSN:0171-2985
Container-title:Immunobiology
language:en
Short-container-title:Immunobiology

Author:

de Córdoba Santiago Rodríguez

Funder

Spanish “Ministerio de Economía y Competitividad”

Fundación Renal Iñigo Alvarez de Toledo

Seventh Framework Programme European Union Project EURenOmics

Autonomous Region of Madrid

Publisher

Elsevier BV

Subject

Hematology,Immunology,Immunology and Allergy

Reference84 articles.

1. Characterization of complement factor H-related (CFHR) proteins in plasma reveals novel genetic variations of CFHR1 associated with atypical hemolytic uremic syndrome;Abarrategui-Garrido;Blood,2009

2. Familial C3 glomerulopathy associated with CFHR5 mutations: clinical characteristics of 91 patients in 16 pedigrees;Athanasiou;Clin. J. Am. Soc. Nephrol.,2011

3. Structural basis for sialic acid-mediated self-recognition by complement factor H;Blaum;Nat. Chem. Biol.,2015

4. Competition between antagonistic complement factors for a single protein on N. meningitidis rules disease susceptibility;Caesar;eLife,2014

5. Complement factor H mutations and gene polymorphisms in haemolytic uraemic syndrome: the C-257T, the A2089G and the G2881T polymorphisms are strongly associated with the disease;Caprioli;Hum. Mol. Genet.,2003

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