Termination of TCR-mediated activation signals is regulated by CrkII-dependent Cbl-mediated ubiquitination and degradation of C3G-Reference-Cited by-同舟云学术

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Termination of TCR-mediated activation signals is regulated by CrkII-dependent Cbl-mediated ubiquitination and degradation of C3G

Published:2023-03 Issue:2 Volume:228 Page:152342
ISSN:0171-2985
Container-title:Immunobiology
language:en
Short-container-title:Immunobiology

Author:

Nath Pulak Ranjan,Anto Nikhil Ponnoor,Braiman Alex,Isakov Noah^ORCID

Funder

Israel Cancer Research Fund

City of Hope

United States-Israel Binational Science Foundation

Israel Academy of Sciences and Humanities

Israel Science Foundation

Ben-Gurion University of the Negev

Publisher

Elsevier BV

Subject

Hematology,Immunology,Immunology and Allergy

Reference39 articles.

1. The two major sites of cbl tyrosine phosphorylation in abl-transformed cells select the crkL SH2 domain;Andoniou;Oncogene,1996

2. The Cbl proto-oncogene product negatively regulates the Src-family tyrosine kinase Fyn by enhancing its degradation;Andoniou;Mol. Cell Biol.,2000

3. Negative regulation of lymphocyte activation and autoimmunity by the molecular adaptor Cbl-b;Bachmaier;Nature,2000

4. The role of crk adaptor proteins in T-cell adhesion and migration;Braiman;Front. Immunol.,2015

5. Activation-induced ubiquitination of the T cell antigen receptor;Cenciarelli;Science,1992

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Conformational and functional regulation of the chicken tumor virus number 10 (CT10) regulator of kinase II (CrkII) adaptor protein by cyclophilin A;Exploration of Immunology;2023-06-30

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