GABAB receptors as potential targets for drugs able to prevent excessive excitatory amino acid transmission in the spinal cord-Reference-Cited by-同舟云学术

GABAB receptors as potential targets for drugs able to prevent excessive excitatory amino acid transmission in the spinal cord

Published:1998-12 Issue:2-3 Volume:362 Page:143-148
ISSN:0014-2999
Container-title:European Journal of Pharmacology
language:en
Short-container-title:European Journal of Pharmacology

Author:

Bonanno Giambattista,Fassio Anna,Sala Roberta,Schmid Giovanna,Raiteri Maurizio

Publisher

Elsevier BV

Subject

Pharmacology

Reference25 articles.

1. Presynaptic γ-hydroxybutyric acid (GHB) and gamma-aminobutyric acidB (GABAB) receptor-mediated release of GABA and glutamate (GLU) in rat thalamic ventrobasal nucleus (VB): a possible mechanism for the generation of absence-like seizures induced by GHB;Banerjee;J. Pharmacol. Exp. Ther.,1995

2. Functional evidence for multiple γ-aminobutyric acidB receptor subtypes in the rat cerebral cortex;Bonanno;J. Pharmacol. Exp. Ther.,1992

3. γ-Aminobutyric acid (GABA) autoreceptors in rat cerebral cortex and spinal cord represent pharmacologically distinct subtypes of the GABAB receptor;Bonanno;J. Pharmacol. Exp. Ther.,1993

4. Multiple GABAB receptors;Bonanno;Trends Pharmacol. Sci.,1993

5. Pharmacologically distinct GABAB receptors that mediate inhibition of GABA and glutamate release in human neocortex;Bonanno;Br. J. Pharmacol.,1997

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