Necrotic Cell Death in C. elegans Requires the Function of Calreticulin and Regulators of Ca2+ Release from the Endoplasmic Reticulum
Author:
Publisher
Elsevier BV
Subject
General Neuroscience
Reference64 articles.
1. Protons activate brain Na+ channel 1 by inducing a conformational change that exposes a residue associated with neurodegeneration;Adams;J. Biol. Chem.,1998
2. Expression of calreticulin in Escherichia coli and identification of its Ca2+ binding domains;Baksh;J. Biol. Chem.,1991
3. Identification of the Zn2+ binding region in calreticulin;Baksh;FEBS Lett.,1995
4. G-alphas-induced neurodegeneration in Caenorhabditis elegans;Berger;J. Neurosci.,1998
5. The genetics of Caenorhabditis elegans;Brenner;Genetics,1974
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