Zebrafish usp3 loss promotes hypoxic tolerance by disrupting deubiquitination of K63-polyubiquitinated hif-1αa-Reference-Cited by-同舟云学术

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Zebrafish usp3 loss promotes hypoxic tolerance by disrupting deubiquitination of K63-polyubiquitinated hif-1αa

Published:2024-04 Issue:2 Volume:3 Page:100245
ISSN:2772-7351
Container-title:Water Biology and Security
language:en
Short-container-title:Water Biology and Security

Author:

Li Jun,Zhou Ziwen,Cai Xiaolian,Song Yanan,Li Zhi,Li Ziyi,Xiao Wuhan,Wang Jing^ORCID

Funder

Chinese Academy of Sciences

National Natural Science Foundation of China

Publisher

Elsevier BV

Reference58 articles.

1. The role of hypoxia-inducible factor post-translational modifications in regulating its localisation, stability, and activity;Albanese;Int. J. Mol. Sci.,2020

2. Sumoylation increases HIF-1alpha stability and its transcriptional activity;Bae;Biochem. Biophys. Res. Commun.,2004

3. SUMOylation of hypoxia-inducible factor-1alpha reduces its transcriptional activity;Berta;Biochem. Biophys. Res. Commun.,2007

4. Deletion of the fih gene encoding an inhibitor of hypoxia-inducible factors increases hypoxia tolerance in zebrafish;Cai;J. Biol. Chem.,2018

5. RSUME, a small RWD-containing protein, enhances SUMO conjugation and stabilizes HIF-1alpha during hypoxia;Carbia-Nagashima;Cell,2007

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