Resistance of the hippocampus in the lactating rat to N-methyl-d-aspartate (NMDA)-mediated excitation is not due to a nonfunctional receptor system
Author:
Publisher
Elsevier BV
Subject
Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience
Reference23 articles.
1. Cortical refractoriness to N-methyl-d,l-aspartic acid (NMA) stimulation in the lactating rat: recovery after pup removal and blockade of progesterone receptors;Abbud;Brain Res.,1993
2. Lactation-induced deficits in NMDA receptor-mediated cortical and hippocampal activation: changes in NMDA receptor gene expression and brainstem activation;Abbud;Brain Res.,1994
3. Lactation inhibits hippocampal and cortical activation of cFos expression by NMDA but not kainate receptor agonists;Abbud;Mol. Cell. Neurosci.,1992
4. Differences in the luteinizing hormone and prolactin response to multiple injections of kainate, as compared to N-methyl-d,l-aspartate, in cycling rats;Abbud;Endocrinology,1991
5. Altered luteinizing hormone and prolactin response to excitatory amino acids during lactation;Abbud;Neuroendocrinology,1993
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