Genotoxic effect of doxorubicin–transferrin conjugate on human leukemia cells

Author:

Szwed Marzena,Jozwiak Zofia

Publisher

Elsevier BV

Subject

Health, Toxicology and Mutagenesis,Genetics

Reference37 articles.

1. Chronic myeloid leukemia with an unusual paraneoplastic syndrome;Sharma;J. Assoc. Physicians India,2012

2. The role of peptide and DNA vaccines in myeloid leukemia immunotherapy;Lin;Cancer Cell. Int.,2013

3. Tyrosine kinase targeted treatment of chronic myelogenous leukemia and other myeloproliferative neoplasms;Bisen;Adv. Exp. Med. Biol.,2013

4. Contribution of BCR-ABL kinase domain mutations to imatinib mesylate resistance in Philadelphia chromosome positive Malaysian chronic myeloid leukemia patients;Elias;Hematol. Rep.,2012

5. Imatinib mesylate (STI571) abrogates the resistance to doxorubicin in human K562 chronic myeloid leukemia cells by inhibition of BCR/ABL kinase-mediated DNA repair;Majsterek;Mutat. Res.,2006

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