Altered imprinting, promoter usage, and expression of insulin-like growth factor-II gene in gestational trophoblastic diseases
Author:
Publisher
Elsevier BV
Subject
Obstetrics and Gynecology,Oncology
Reference29 articles.
1. Expression of the p53 gene and apotosis in gestational trophoblastic disease;Halperin;Placenta,2000
2. Telomerase activity in complete hydatidiform mole;Bae;Am J Obstet Gynecol,1999
3. Differential expression of p57KIP2, a maternally imprinted cdk inhibitor, in normal human placenta and gestational trophoblastic disease;Chilosi;Lab Invest,1998
4. Frequent loss of imprinting of the H19 and IGF-II genes in ovarian tumors;Kim;Am J Med Genet,1998
5. Alterations in promoter usage and expression levels of insulin-like growth factor-II and H19 genes in cervical carcinoma exhibiting biallelic expression of IGF-II;Kim;Biochim Biophys Acta,2002
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1. Lentivirus-mediated RNA interference targeting the H19 gene inhibits cell proliferation and apoptosis in human choriocarcinoma cell line JAR;BMC Cell Biology;2013-05-27
2. Loss of imprinting and aberrant methylation of IGF2 in placentas from pregnancies complicated with fetal growth restriction;International Journal of Molecular Medicine;2011-07-19
3. Assisted reproductive technologies do not increase risk of abnormal methylation of PEG1/MEST in human early pregnancy loss;Fertility and Sterility;2011-07
4. Increased insulin resistance and C-reactive protein in women with complete hydatidiform mole;Gynecological Endocrinology;2011-01-04
5. Epigenetics and the placenta;Human Reproduction Update;2010-10-19
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