The zellweger syndrome: Deficient conversion of docosahexaenoic acid (22:6(n−3)) to eicosapentaenoic acid (20:5(n−3)) and normal Δ4-desaturase activity in cultured skin fibroblasts
Author:
Publisher
Elsevier BV
Subject
Endocrinology,Biochemistry,Biophysics
Reference32 articles.
1. Retroconversion of docosahexaenoic acid in the rat
2. Metabolism and effects on platelet function of the purified eicosapentaenoic and docosahexaenoic acids in humans.
3. Metabolism of 4,7,10,13,16-Docosapentaenoic Acid in the Essential Fatty Acid-deficient Rat
4. Retroconversion of polyunsaturated fatty acids in vivo by partial degradation and hydrogenation
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1. Low colostrum intake results in potential accumulation of peroxisome lipid substrates in vaginal tissue of 3-week-old gilts;Biology Open;2023-08-11
2. Retroconversion is a minor contributor to increases in eicosapentaenoic acid following docosahexaenoic acid feeding as determined by compound specific isotope analysis in rat liver;Nutrition & Metabolism;2017-11-28
3. Metabolic fate of docosahexaenoic acid (DHA; 22:6n-3) in human cells: direct retroconversion of DHA to eicosapentaenoic acid (20:5n-3) dominates over elongation to tetracosahexaenoic acid (24:6n-3);FEBS Letters;2016-09
4. Molecular Species of Phospholipids with Very Long Chain Fatty Acids in Skin Fibroblasts of Zellweger Syndrome;Lipids;2013-10-13
5. Phosphatidyl ethanolamine with increased polyunsaturated fatty acids in compensation for plasmalogen defect in the Zellweger syndrome brain;Neuroscience Letters;2009-01
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