Lecithin:cholesterol acyltransferase in familial HDL deficiency (Tangier disease)
Author:
Publisher
Elsevier BV
Subject
Endocrinology,Biochemistry,Biophysics
Reference36 articles.
1. Clinical, biochemical, and genetic features in familial disorders of high density lipoprotein deficiency.
2. Tangier Disease
3. The Inheritance of High Density Lipoprotein Deficiency (Tangier Disease)*
4. Tangier Disease
5. Identification of lipoprotein families in a variant of human plasma apolipoprotein A deficiency
Cited by 25 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Treatment of hypertriglyceridemia by two diets rich either in unsaturated fatty acids or in carbohydrates: effects on lipoprotein subclasses, lipolytic enzymes, lipid transfer proteins, insulin and leptin;International Journal of Obesity;2000-10
2. Cholesterol efflux from normal and Tangier disease fibroblasts into normal, high-density lipoprotein-deficient, and apolipoprotein E-deficient plasmas;Metabolism;2000-06
3. Apolipoprotein A-I(R151C)Paris is defective in activation of lecithin:cholesterol acyltransferase but not in initial lipid binding, formation of reconstituted lipoproteins, or promotion of cholesterol efflux;Journal of Molecular Medicine;1999-08
4. Multiple dysfunctions of two apolipoprotein A-I variants, apoA-I(R160L)Oslo and apoA-I(P165R), that are associated with hypoalphalipoproteinemia in heterozygous carriers;Journal of Lipid Research;1999-03
5. Lipid-free apolipoprotein (apo) A-I is converted into alpha-migrating high density lipoproteins by lipoprotein-depleted plasma of normolipidemic donors and apo A-I-deficient patients but not of Tangier disease patients;Atherosclerosis;1998-05
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