Affinity Proteomics for Phosphatase Interactions in Atrial Fibrillation∗-Reference-Cited by-同舟云学术

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Affinity Proteomics for Phosphatase Interactions in Atrial Fibrillation∗

Published:2015-01 Issue:2 Volume:65 Page:174-176
ISSN:0735-1097
Container-title:Journal of the American College of Cardiology
language:en
Short-container-title:Journal of the American College of Cardiology

Author:

Barallobre-Barreiro Javier,Mayr Manuel

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine

Reference17 articles.

1. The clinical profile and pathophysiology of atrial fibrillation: relationships among clinical features, epidemiology, and mechanisms;Andrade;Circ Res,2014

2. Diastolic dysfunction and arrhythmias caused by overexpression of CaMKIIδ(C) can be reversed by inhibition of late Na(+) current;Sossalla;Basic Res Cardiol,2011

3. A dynamic pathway for calcium-independent activation of CaMKII by methionine oxidation;Erickson;Cell,2008

4. Calmodulin kinase II-mediated sarcoplasmic reticulum Ca2+ leak promotes atrial fibrillation in mice;Chelu;J Clin Invest,2009

5. Calmodulin kinase II inhibition prevents arrhythmic activity induced by alpha and beta adrenergic agonists in rabbit pulmonary veins;Lo;Eur J Pharmacol,2007

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Protein Phosphatase Signaling in Cardiac Myocytes;Cardiovascular Signaling in Health and Disease;2022

2. Proteomics and transcriptomics in atrial fibrillation;Herzschrittmachertherapie + Elektrophysiologie;2018-01-09

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