Longitudinal Change in Galectin-3 and Incident Cardiovascular Outcomes-Reference-Cited by-同舟云学术

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Longitudinal Change in Galectin-3 and Incident Cardiovascular Outcomes

Published:2018-12 Issue:25 Volume:72 Page:3246-3254
ISSN:0735-1097
Container-title:Journal of the American College of Cardiology
language:en
Short-container-title:Journal of the American College of Cardiology

Author:

Ghorbani Anahita,Bhambhani Vijeta,Christenson Robert H.,Meijers Wouter C.,de Boer Rudolf A.,Levy Daniel,Larson Martin G.,Ho Jennifer E.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine

Reference32 articles.

1. Galectin-3: a novel mediator of heart failure development and progression;de Boer;Eur J Heart Fail,2009

2. Galectin-3 in cardiac remodeling and heart failure;de Boer;Curr Heart Fail Rep,2010

3. Galectin-3 marks activated macrophages in failure-prone hypertrophied hearts and contributes to cardiac dysfunction;Sharma;Circulation,2004

4. N-acetyl-seryl-aspartyl-lysyl-proline prevents cardiac remodeling and dysfunction induced by galectin-3, a mammalian adhesion/growth-regulatory lectin;Liu;Am J Physiol Heart Circ Physiol,2009

5. Genetic and pharmacological inhibition of galectin-3 prevents cardiac remodeling by interfering with myocardial fibrogenesis;Yu;Circ Heart Fail,2013

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2. Global characterization of myeloid cells in the human failing heart;Science Bulletin;2024-05

3. Galectin‐3, Metabolic Risk, and Incident Heart Failure: The ARIC Study;Journal of the American Heart Association;2024-03-19

4. Effects of harvesting and an invasive mussel on intertidal rocky shore communities based on historical and spatial comparisons;PLOS ONE;2024-02-08

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