Ventricular Unloading, Tissue Angiotensin II, Matrix Modulation, and Function During Left Ventricular Assist Device Support⁎⁎Editorials published in the Journal of the American College of Cardiologyreflect the views of the authors and do not necessarily represent the views of JACCor the American College of Cardiology.
Author:
Publisher
Elsevier BV
Subject
Cardiology and Cardiovascular Medicine
Reference10 articles.
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2. Left ventricular assist device as destination for patients undergoing intravenous inotropic therapy: a subset analysis from REMATCH (Randomized Evaluation of Mechanical Assistance in Treatment of Chronic Heart Failure);Stevenson;Circulation,2004
3. Remodeling of the myocardium and potential targets in the collagen degradation and synthesis pathways;Jugdutt;Curr Drug Targets Cardiovasc Haematol Disord,2003
4. Gene expression in fibroblasts and fibrosis: involvement in cardiac hypertrophy;Manabe;Circ Res,2002
5. Mechanical unloading during left ventricular assist device support increases left ventricular collagen cross-linking and myocardial stiffness;Klotz;Circulation,2005
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3. Effect of Neurohormonal Blockade Drug Therapy on Outcomes and Left Ventricular Function and Structure After Left Ventricular Assist Device Implantation;The American Journal of Cardiology;2016-06
4. The paradox of left ventricular assist device unloading and myocardial recovery in end-stage dilated cardiomyopathy: implications for heart failure in the elderly;Heart Failure Reviews;2012-01-18
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