PCSK9 Inhibitors for Homozygous Familial Hypercholesterolemia
Author:
Publisher
Elsevier BV
Subject
Cardiology and Cardiovascular Medicine
Reference11 articles.
1. Familial hypercholesterolemia: identification of a defect in the regulation of 3-hydroxy-3-methylglutaryl coenzyme A reductase activity associated with overproduction of cholesterol;Goldstein;Proc Natl Acad Sci U S A,1973
2. Familial hypercholesterolemia: defective binding of lipoproteins to cultured fibroblasts associated with impaired regulation of 3-hydroxy-3-methylglutaryl coenzyme A reductase activity;Brown;Proc Natl Acad Sci U S A,1974
3. Improved cardiovascular outcomes following temporal advances in lipid-lowering therapy in a genetically-characterised cohort of familial hypercholesterolaemia homozygotes;Thompson;Atherosclerosis,2015
4. Atherosclerosis in cholesterol-fed rabbits and in homozygous and heterozygous LDL receptor-deficient humans;Thompson;Atherosclerosis,2018
5. Lipoprotein apheresis in the management of familial hypercholesterolaemia: historical perspective and recent advances;Stefanutti;Curr Atheroscler Rep,2015
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1. Proprotein convertase subtilisin/kexin type 9-inhibition across different patient populations;Current Opinion in Lipidology;2024-03-27
2. Failure of lipid control by PCSK9 inhibitors in compound heterozygous familial hypercholesterolemia complicated with premature myocardial infarction: A case report;Clinical Case Reports;2024-03
3. New Oral PCSK9 Inhibitor: “MK-0616”;Cardiology in Review;2024-01-29
4. Does Genotype Affect the Efficacy of PCSK9 Inhibitors in the Treatment of Familial Hypercholesterolemia?;Cardiovascular Drugs and Therapy;2023-08-23
5. How Genetic Variants in Children with Familial Hypercholesterolemia Not Only Guide Detection, but Also Treatment;Genes;2023-03-07
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