AHR activation by tryptophan—Pathogenic hallmark of Th17-mediated inflammation in eosinophilic fasciitis, eosinophilia–myalgia-syndrome and toxic oil syndrome?-Reference-Cited by-同舟云学术

AHR activation by tryptophan—Pathogenic hallmark of Th17-mediated inflammation in eosinophilic fasciitis, eosinophilia–myalgia-syndrome and toxic oil syndrome?

Published:2010-02 Issue:2 Volume:128 Page:154-155
ISSN:0165-2478
Container-title:Immunology Letters
language:en
Short-container-title:Immunology Letters

Author:

Rieber Nikolaus,Belohradsky Bernd H.

Publisher

Elsevier BV

Subject

Immunology,Immunology and Allergy

Reference11 articles.

1. The aryl hydrocarbon receptor links TH17-cell-mediated autoimmunity to environmental toxins;Veldhoen;Nature,2008

2. Control of Treg and TH17 cell differentiation by the aryl hydrocarbon receptor;Quintana;Nature,2008

3. The search for endogenous activators of the aryl hydrocarbon receptor;Nguyen;Chem Res Toxicol,2008

4. Modulation of Th17 development and function by activation of the aryl hydrocarbon receptor—the role of endogenous ligands;Stockinger;Eur J Immunol,2009

5. Natural agonists for aryl hydrocarbon receptor in culture medium are essential for optimal differentiation of Th17 T cells;Veldhoen;J Exp Med,2009

Cited by 13 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Multi-omics approaches for the understanding of therapeutic mechanism for Huang-Qi-Long-Dan Granule against ischemic stroke;Pharmacological Research;2024-07

2. L-Tryptophan activates the aryl hydrocarbon receptor and induces cell cycle arrest in porcine trophectoderm cells;Theriogenology;2021-09

3. Eosinophilia-Myalgia Syndrome;Drug-induced Neurological Disorders;2021

4. Chemical exposure-induced systemic fibrosing disorders: Novel insights into systemic sclerosis etiology and pathogenesis;Seminars in Arthritis and Rheumatism;2020-12

5. Gut dysbiosis, leaky gut, and intestinal epithelial proliferation in neurological disorders: towards the development of a new therapeutic using amino acids, prebiotics, probiotics, and postbiotics;Reviews in the Neurosciences;2019-01-28