Why does chronic inflammation persist: An unexpected role for fibroblasts
Author:
Publisher
Elsevier BV
Subject
Immunology,Immunology and Allergy
Reference15 articles.
1. Interferon‐β mediates stromal cell rescue of T cells from apoptosis
2. Persistent Induction of the Chemokine Receptor CXCR4 by TGF-β1 on Synovial T Cells Contributes to Their Accumulation Within the Rheumatoid Synovium
3. Fibroblasts regulate the switch from acute resolving to chronic persistent inflammation
4. Ectopic expression of the B cell-attracting chemokine BCA-1 (CXCL13) on endothelial cells and within lymphoid follicles contributes to the establishment of germinal center-like structures in Sj�gren's syndrome
5. Rheumatoid fibroblast-like synoviocytes overexpress the chemokine stromal cell-derived factor 1 (CXCL12), which supports distinct patterns and rates of CD4+ and CD8+ T cell migration within synovial tissue
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