The Greater Susceptibility of North Ronaldsay Sheep Compared with Cambridge Sheep to Copper-induced Oxidative Stress, Mitochondrial Damage and Hepatic Stellate Cell Activation

Author:

Haywood S.,Simpson D.M.,Ross G.,Beynon R.J.

Publisher

Elsevier BV

Subject

General Veterinary,Pathology and Forensic Medicine

Reference27 articles.

1. Yeast contains a non-proteinacious pool of copper in the mitochondrial matrix;Cobine;Journal of Biological Chemistry,2004

2. Dincer, Z., 1994. Copper toxicity in sheep: studies on copper chelation by ammonium tetrathiomolybdate (TTM) and metallothionein. PhD Thesis, University of Liverpool.

3. GSH transport in mitochondria: defense against TNF-induced oxidative stress and alcohol-induced defect;Fernández-Checa;American Journal of the Physiological Society,1997

4. New insights into hepatic stellate cell activation: the simple becomes complex;Friedman;American Journal of Physiology. Gastrointestial and Liver Physiology,2000

5. Cellular mechanisms of toxicity and tolerance in the copper-loaded rat. II. Pathogenesis of copper toxicity in the liver;Fuentealba;Experimental and Molecular Pathology,1989

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