Mitochondria DNA mutations cause sex-dependent development of hypertension and alterations in cardiovascular function-Reference-Cited by-同舟云学术

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Mitochondria DNA mutations cause sex-dependent development of hypertension and alterations in cardiovascular function

Published:2015-02 Issue:3 Volume:48 Page:405-412
ISSN:0021-9290
Container-title:Journal of Biomechanics
language:en
Short-container-title:Journal of Biomechanics

Author:

Golob Mark J.,Tian Lian,Wang Zhijie,Zimmerman Todd A.,Caneba Christine A.,Hacker Timothy A.,Song Guoqing,Chesler Naomi C.

Funder

NIH

T32

Publisher

Elsevier BV

Subject

Rehabilitation,Biomedical Engineering,Orthopedics and Sports Medicine,Biophysics

Reference53 articles.

1. Consistent biomechanical phenotyping of common carotid arteries from seven genetic, pharmacological, and surgical mouse models;Bersi;Ann. Biomed. Eng.,2014

2. Distinct loading conditions reveal various patterns of right ventricular adaptation;Borgdorff;Am. J. Physiol.: Heart Circ. Physiol.,2013

3. Ablation of the cardiac-specific gene leucine-rich repeat containing 10 (lrrc10) results in dilated cardiomyopathy;Brody;PloS One,2012

4. A novel murine model of severe pulmonary arterial hypertension;Ciuclan;Am. J. Respir. Crit. Care Med.,2011

5. Age-dependent cardiomyopathy in mitochondrial mutator mice is attenuated by overexpression of catalase targeted to mitochondria;Dai;Aging Cell,2010

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2. mt-tRNAs in the polymerase gamma mutant heart;The Journal of Cardiovascular Aging;2023-10-18

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4. Mito-SiPE is a sequence-independent and PCR-free mtDNA enrichment method for accurate ultra-deep mitochondrial sequencing;Communications Biology;2022-11-19

5. Mito-SiPE: A sequence-independent, PCR-free mitochondrial DNA enrichment method for ultra-deep sequencing that minimises amplification and alignment artefacts for the analysis of mitochondrial heteroplasmy/variation;2022-10-17

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