Epigenetic modification of the gene for the vitamin B12 chaperone MMACHC can result in increased tumorigenicity and methionine dependence-Reference-Cited by-同舟云学术

Epigenetic modification of the gene for the vitamin B12 chaperone MMACHC can result in increased tumorigenicity and methionine dependence

Published:2009-04 Issue:4 Volume:96 Page:261-267
ISSN:1096-7192
Container-title:Molecular Genetics and Metabolism
language:en
Short-container-title:Molecular Genetics and Metabolism

Author:

Loewy Amanda D.,Niles Kirsten M.,Anastasio Natascia,Watkins David,Lavoie Josée,Lerner-Ellis Jordan P.,Pastinen Tomi,Trasler Jacquetta M.,Rosenblatt David S.

Publisher

Elsevier BV

Subject

Endocrinology,Genetics,Molecular Biology,Biochemistry,Endocrinology, Diabetes and Metabolism

Reference33 articles.

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3. The metabolic defect of methionine dependence occurs frequently in human tumor cell lines;Mecham;Biochem. Biophys. Res. Commun.,1983

4. High in vivo rates of methionine biosynthesis in transformed human and malignant rat cells auxotrophic for methionine;Hoffman;Proc. Natl. Acad. Sci. USA,1976

5. Methionine metabolism defect in cells transfected with an activated HRAS1 oncogene;Vanhamme;Exp. Cell Res.,1987

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