Distinct roles for matrix metalloproteinase-2 and α4 integrin in autoimmune T cell extravasation and residency in brain parenchyma during experimental autoimmune encephalomyelitis

Author:

Graesser Donnasue,Mahooti Sepi,Madri Joseph A

Publisher

Elsevier BV

Subject

Neurology (clinical),Neurology,Immunology,Immunology and Allergy

Reference29 articles.

1. Surface expression of α4 integrin by CD4 T cells is required for their entry into brain parenchyma;Baron;J. Exp. Med.,1993

2. VLA-4 expression on peripheral blood lymphocytes is downregulated after treatment of multiple sclerosis with interferon beta;Calabresi;Neurology,1997

3. Reversal of experimental autoimmune encephalomyelitis with a hydroxymate inhibitor of matrix metalloproteases;Gijbels;J. Clin. Invest.,1994

4. The interrelationship of α4 integrin and matrix metalloproteinase-2 in the pathogenesis of experimental autoimmune encephalomyelitis;Graesser;Lab. Invest.,1998

5. T cells with two functional antigen-specific receptors;Hardardottir;Proc. Natl. Acad. Sci. USA,1995

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