High innate production of interleukin-10 and tumor necrosis factor-α contributes to susceptibility for non-paraneoplastic Lambert–Eaton myasthenic syndrome
Author:
Publisher
Elsevier BV
Subject
Clinical Neurology,Neurology,Immunology,Immunology and Allergy
Cited by 6 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Lambert-Eaton myasthenic syndrome;Handbook of Clinical Neurology;2024
2. Multifocal motor neuropathy is not associated with altered innate immune responses to endotoxin;Journal of the Neurological Sciences;2023-08
3. Hereditary and Autoimmune Myasthenias;Emery and Rimoin's Principles and Practice of Medical Genetics;2013
4. Lambert-Eaton myasthenic syndrome: A long-term survivor after multimodality therapy for small cell lung cancer;The Journal of the Japanese Associtation for Chest Surgery;2006
5. HLA and smoking in prediction and prognosis of small cell lung cancer in autoimmune Lambert–Eaton myasthenic syndrome;Journal of Neuroimmunology;2005-02
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