Immunologic mechanisms in granulomatous lung disease

Author:

Newman Lee S

Publisher

Elsevier BV

Subject

Pharmacology

Reference19 articles.

1. Alveolar macrophages from patients with beryllium disease and sarcoidosis express increased levels of mRNA for TNF-α and IL-6 but not IL-1β;Bost;Am. J. Respir. Cell Mol. Biol.,1994

2. Identification of pathogenic T cells in patients with beryllium-induced lung disease;Fontenot;J. Immunol.,1999

3. Expansions of T cell subsets expressing particular T cell receptor variable regions in chronic beryllium disease;Fontenot;Am. J. Respir. Cell Mol. Biol.,1998

4. TCR expression of activated T cell clones in the lungs of patients with pulmonary sarcoidosis;Forrester;J. Immunol.,1994

5. Biased expression of individual T cell receptor V gene segments in CD4+ and CD8+ human peripheral blood T lymphocytes;Grunewald;Eur. J. Immunol.,1991

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1. Risk of beryllium sensitization in a low-exposed former nuclear weapons cohort from the cold war era;American Journal of Industrial Medicine;2010-10-28

2. Increased levels of nerve growth factor in the airways of patients with sarcoidosis;Journal of Internal Medicine;2008-11

3. Mechanisms of Granuloma Formation;Lung Biology in Health and Disease;2005-12

4. Beryllium as a Model for Sarcoidosis;Lung Biology in Health and Disease;2005-12

5. Why the Search for the Etiology of Sarcoidosis Should Continue;Lung Biology in Health and Disease;2005-12

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