Molecular Mechanisms of Hepatic Steatosis and Insulin Resistance in the AGPAT2-Deficient Mouse Model of Congenital Generalized Lipodystrophy-Reference-Cited by-同舟云学术

Molecular Mechanisms of Hepatic Steatosis and Insulin Resistance in the AGPAT2-Deficient Mouse Model of Congenital Generalized Lipodystrophy

Published:2009-02 Issue:2 Volume:9 Page:165-176
ISSN:1550-4131
Container-title:Cell Metabolism
language:en
Short-container-title:Cell Metabolism

Author:

Cortés Víctor A.,Curtis David E.,Sukumaran Suja,Shao Xinli,Parameswara Vinay,Rashid Shirya,Smith Amy R.,Ren Jimin,Esser Victoria,Hammer Robert E.,Agarwal Anil K.,Horton Jay D.,Garg Abhimanyu

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Physiology

Reference62 articles.

1. Congenital generalized lipodystrophy: significance of triglyceride biosynthetic pathways;Agarwal;Trends Endocrinol. Metab.,2003

2. Genetic disorders of adipose tissue development, differentiation, and death;Agarwal;Annu. Rev. Genomics Hum. Genet.,2006

3. AGPAT2 is mutated in congenital generalized lipodystrophy linked to chromosome 9q34;Agarwal;Nat. Genet.,2002

4. Phenotypic and genetic heterogeneity in congenital generalized lipodystrophy;Agarwal;J. Clin. Endocrinol. Metab.,2003

5. Functional characterization of human 1-acylglycerol-3-phosphate acyltransferase isoform 8: cloning, tissue distribution, gene structure and enzymatic activity;Agarwal;Arch. Biochem. Biophys.,2006

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