Preserving Insulin Secretion in Diabetes by Inhibiting VDAC1 Overexpression and Surface Translocation in β Cells-Reference-Cited by-同舟云学术

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Preserving Insulin Secretion in Diabetes by Inhibiting VDAC1 Overexpression and Surface Translocation in β Cells

Published:2019-01 Issue:1 Volume:29 Page:64-77.e6
ISSN:1550-4131
Container-title:Cell Metabolism
language:en
Short-container-title:Cell Metabolism

Author:

Zhang Enming,Mohammed Al-Amily Israa,Mohammed Sarheed,Luan Cheng,Asplund Olof,Ahmed Meftun,Ye Yingying,Ben-Hail Danya,Soni Arvind,Vishnu Neelanjan,Bompada Pradeep,De Marinis Yang,Groop Leif,Shoshan-Barmatz Varda,Renström Erik,Wollheim Claes B.,Salehi Albert

Funder

Sweden, Öresund Diabetes Academy

Mats Paulsson Foundation

Forget foundation

Bo and Kerstin Hjelt foundation

Lund University

Crafoord Foundation

Israel Science Foundation

Sima and Philip Needleman research

ERC Advanced Researcher

Knut and Alice Wallenberg Foundation

Swedish Research Council

Swedish Diabetes Association

Diabetes Wellness Sweden Foundation

ALF

Strategic Research Area Exodiab

Strategic Research LUDC-IRC

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Physiology

Reference54 articles.

1. Voltage-dependent anion channels (VDAC) in the plasma membrane play a critical role in apoptosis in differentiated hippocampal neurons but not in neural stem cells;Akanda;Cell Cycle,2008

2. Mitochondrial proteome analysis reveals altered expression of voltage dependent anion channels in pancreatic beta-cells exposed to high glucose;Ahmed;Islets,2010

3. Functional and morphological alterations of mitochondria in pancreatic beta cells from type 2 diabetic patients;Anello;Diabetologia,2005

4. VDAC1 cysteine residues: topology and function in channel activity and apoptosis;Aram;Biochem. J.,2010

5. Metformin for diabetes prevention: insights gained from the diabetes prevention program/diabetes prevention program outcomes study;Aroda;Diabetologia,2017

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