Loss of TAZ Boosts PPARγ to Cope with Insulin Resistance
Author:
Funder
JDRF
German Research Foundation
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Physiology
Reference11 articles.
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3. TAZ is a negative regulator of PPARγ activity in adipocytes and TAZ deletion improves insulin sensitivity and glucose tolerance;El Ouarrat;Cell Metab.,2020
4. TAZ, a transcriptional modulator of mesenchymal stem cell differentiation;Hong;Science,2005
5. Interplay between YAP/TAZ and Metabolism;Koo;Cell Metab.,2018
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