Glucagon promotes increased hepatic mitochondrial oxidation and pyruvate carboxylase flux in humans with fatty liver disease-Reference-Cited by-同舟云学术

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Glucagon promotes increased hepatic mitochondrial oxidation and pyruvate carboxylase flux in humans with fatty liver disease

Published:2024-08 Issue: Volume: Page:
ISSN:1550-4131
Container-title:Cell Metabolism
language:en
Short-container-title:Cell Metabolism

Author:

Petersen Kitt Falk,Dufour Sylvie,Mehal Wajahat Z.,Shulman Gerald I.^ORCID

Publisher

Elsevier BV

Reference61 articles.

1. AGA technical review on nonalcoholic fatty liver disease;Sanyal;Gastroenterology,2002

2. Mechanisms and disease consequences of nonalcoholic fatty liver disease;Loomba;Cell,2021

3. Predictors of all-cause mortality and liver-related mortality in patients with non-alcoholic fatty liver disease (NAFLD);Stepanova;Dig. Dis. Sci.,2013

4. Excessive hepatic mitochondrial TCA cycle and gluconeogenesis in humans with nonalcoholic fatty liver disease;Sunny;Cell Metab.,2011

5. Adaptation of hepatic mitochondrial function in humans with non-alcoholic fatty liver is lost in steatohepatitis;Koliaki;Cell Metab.,2015

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